Is a zero-plaque heart scan on keto proof of safety? Discover how plaque latency explains a CAC of 0 despite extreme LDL, and why medical guidelines still urge caution.
If you are a competitive endurance athlete over the age of 65, you are likely living through a frustrating biological mystery. You still have the drive. You are still putting in the long miles on your bike or hitting the trail for your morning run. But despite your hard work, your body seems to be changing in ways that don't feel fair.
Imagine you have just started a new way of eating. You have cut out bread, pasta, and sugar. Instead, you are eating more healthy fats, like steak, eggs, and butter. You feel better than you have in years. Your "sugar levels" are great, your blood pressure is low, and you have plenty of energy. In your mind, you are "metabolically perfect." This means your body is doing a great job of handling energy and keeping you fit.
Imagine a doctor sitting in a dark room, peering through a powerful microscope at a tiny blob of yellow fat. For almost a hundred years, doctors looked at these blobs and thought they had found the "bad guy" causing heart disease. They called it cholesterol, and they believed that simply having too much of this "yellow grease" in your blood was the reason pipes in the body got clogged.
In a world of "biohacking" and rapid weight-loss trends, two dietary paradigms often clash: the Low-Fat Whole Food Plant-Based (WFPB) diet and the high-protein/high-fat Paleo/Ketogenic diets. While both claim to improve health, the scientific literature—specifically long-term clinical trials and centenarian studies—reveals a clear distinction between short-term metabolic shifts and long-term disease reversal.
What do people on social media mean by “ketosis,” and does it automatically mean “healthier”?
Ketosis simply means your blood ketones (often β-hydroxybutyrate, βHB) are elevated because carbohydrate availability is low (fasting, very-low-carb diets) or because you ingested ketones.
Dr. Loren Cordain vs. Dr. T. Colin Campbell
Original context: Public nutrition debate from the mid-2000s reflecting two competing paradigms: evolutionary biology versus whole-diet epidemiology.
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